![]() The disease progression will depend on the type and location of these abnormal vessels, the possible occurrence of retinal uplifts, bleeding, and the response to treatment. The abnormal vessels are fragile and allow the serum to diffuse, which can cause serious detachment and lead to hemorrhage. The second form is a wet or “exudative” or “neovascular” form. The aim of the non-exudative AMD treatment is to delay the loss of visual function. For the atrophic form, only nutritional supplementation is currently given since no therapies have been shown to be effective. It is usually classed into one of two forms. The first warning signs-deformation of straight lines, abrupt decrease in visual acuity or contrasts, dark central spot-should lead to prompt consultation at an ophthalmologist. Finally, there is the development of atrophy (“dry AMD”) or of so-called new vessels in the macular region that seem to play a very important role in the complications associated with “wet AMD”. Subsequently, increased cell death is observed along with inflammation, which is activated by the accumulation of extracellular and intracellular debris. AMD is characterized by different key steps such as oxidative damage, lipofuscin accumulation, and impaired activity or function of the retinal pigmented epithelium (RPE). ![]() This disease is characterized by damage to the macula, the central area of the retina, which allows for fine or central vision. Among the degenerative pathologies linked to age, eye diseases represent a significant part, and in particular age-related macular degeneration (AMD), which is one of the main causes of deterioration in vision in adults aged 55 and older in developed countries with an obvious negative impact on quality of life. ![]()
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